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Nonclassical IL-1 beta secretion stimulated by P2X7 receptors is dependent on inflammasome activation and correlated with exosome release in murine macrophages. Colony-stimulating factor 1 receptor signaling is necessary for microglia viability, unmasking a microglia progenitor cell in the adult brain. Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model. MFG-E8 mediates primary phagocytosis of viable neurons during neuroinflammation. Severity of gliosis in Pick's disease and frontotemporal lobar degeneration: tau-positive glia differentiate these disorders. Schofield, E., Kersaitis, C., Shepherd, C.E., Kril, J.J. Promoters and serotypes: targeting of adeno-associated virus vectors for gene transfer in the rat central nervous system in vitro and in vivo. Shevtsova, Z., Malik, J.M., Michel, U., Bahr, M. Reactive glia not only associates with plaques but also parallels tangles in Alzheimer's disease. Tau-66: evidence for a novel tau conformation in Alzheimer's disease. Extracellular vesicles: biology and emerging therapeutic opportunities. Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner. Debris clearance by microglia: an essential link between degeneration and regeneration. Identification of preclinical Alzheimer's disease by a profile of pathogenic proteins in neurally derived blood exosomes: a case-control study. Exosome-associated tau is secreted in tauopathy models and is selectively phosphorylated in cerebrospinal fluid in early Alzheimer disease. Potential contribution of exosomes to the prion-like propagation of lesions in Alzheimer's disease. Distinct tau prion strains propagate in cells and mice and define different tauopathies. Neuronal activity regulates extracellular tau in vivo. Intracerebral injection of preformed synthetic tau fibrils initiates widespread tauopathy and neuronal loss in the brains of tau transgenic mice. Trans-synaptic spread of tau pathology in vivo. Propagation of tau pathology in a model of early Alzheimer's disease. Human P301L-mutant tau expression in mouse entorhinal-hippocampal network causes tau aggregation and presynaptic pathology but no cognitive deficits.
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Synthetic tau fibrils mediate transmission of neurofibrillary tangles in a transgenic mouse model of Alzheimer's-like tauopathy. Differential induction and spread of tau pathology in young PS19 tau transgenic mice following intracerebral injections of pathological tau from Alzheimer's disease or corticobasal degeneration brains. A novel in vivo model of tau propagation with rapid and progressive neurofibrillary tangle pathology: the pattern of spread is determined by connectivity, not proximity. Transmission and spreading of tauopathy in transgenic mouse brain. Neuropathological stageing of Alzheimer-related changes.